Resistance of the target islet tissue to autoimmune destruction contributes to genetic susceptibility in Type 1 diabetes
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* Corresponding author: Nora Sarvetnick noras@scripps.edu
1 Department of Immunology, The Scripps Research Institute, La Jolla, California, USA
2 Department of Molecular Biology and the Skaggs Institute for Chemical Biology, The Scripps Research Institute, La Jolla, California, USA
3 Centre for Diabetes and Metabolic Medicine, Institute of Cell and Molecular Sciences, Barts and the London Queen Mary's School of Medicine and Dentistry, London, UK
Biology Direct 2007, 2:5 doi:10.1186/1745-6150-2-5
Published: 25 January 2007Additional files
Additional File 1:
Chimerism data for the experiment in Figure 3D. This figure shows the %Thy1.1+ cells within CD4 and CD8 T cell populations for secondary lymphoid organs in irradiated Idd9 (circles) and NOD (filled diamonds) recipients. No difference in %Thy1.1 (donor) cells between Idd9 congenic and NOD recipients is observed.
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